One big issue in the making of products from cattle is that hormones are being added to dairy cattle in order to make them grow faster and produce more milk. The U.S government stated that the hormones are safe and do not lead to any cancerous bacteria. Either way, hormones are a risk to the younger generation, where many kids are now eating lost of fast food. This can cause a rapid growth in a child leading to further maturing problems.
Personally, I know a young boy who loved to eat lots of hot dogs. He was ten years old at that time and suddenly he began maturing way to fast. At first, his mother thought it was just that age where he's going into teenage hood . Unfortunately he began growing facial hair at the age of eleven. Something was wrong, and it was. Do to him eating hot dogs so often, the hormones implanted in the meat caused a rapid development in his system. Automatically the doctor demanded , that he no longer eats any of the products for a while. Even if we look at teenagers now, they look so mature, way beyond their age. We all should be concerned with the choices in food we make.
There is many natural and organic markets opening up. It might be a bit expensive, but when it comes to our health we shouldn't be careless. Simple things as reading the labels, finding out where this product has been made can really improve our way of eating. Also, researching sites where people gather together to ban hormones being implanted in cattle, and support. The more people take charge, the more change will come. People really need to realize that hormones implanted in cattle means, hormones implanted in humans. Again, take a stand read the label and do not continue to support the unsafe way of making our everyday meat products.
Sources to use :
1.) Source:
Nutrition Research Reviews. Dec2002, Vol. 15 Issue 2, p293-314. 22p.
Document Type: Article
Authors:
Hugh Galbraith1 -Methods of production vary and include the use of hormonal compounds (‘
hormones’) to increase growth and lean tissue with reduced fat deposition in cattle.
- The use of
hormones as production aids is permitted in North American countries but is no longer allowed in the European Union (EU), which also prohibits the importation of beef and its products derived from
hormone-treated cattle. These actions have resulted in a trade dispute between the two trading blocs.
-Particular considerations include the low quantities of hormonal compounds consumed in
meat products and their relationships to endogenous production particularly in prepubertal children, enterohepatic inactivation, cellular receptor- and non-receptor-mediated effects and potential for interference with growth, development and physiological function in consumers.
-Classical oestrogen-receptor mediation is considered to stimulate proliferation in cells maintaining receptivity. Mathematical models describing quantitative relationships between consumption of small amounts of oestrogens in
meat in addition to greater concentrations from endogenous production, chemical stoichiometry at cellular level and human pathology have not been developed.
-The other
hormones, although generally less well researched, are similarly subject to a range of tests to determine potentially adverse effects.
-The present review considers the
hormone issue in the context of current international social methodology and regulation, recent advances in knowledge of biological activity of
hormones and current status of science-based evaluation of food safety and risk for human consumers.
2.)Source :
Can hormones contained in mothers’ milk account for the beneficial effect of breast-feeding on obesity in children?
Savino, Francesco, et al. "Can Hormones Contained In Mothers’ Milk Account For The Beneficial Effect Of Breast-Feeding On Obesity In Children?." Clinical Endocrinology 71.6 (2009): 757-765. Academic Search Complete. Web. 31 Oct. 2013.
-Nutrition and growth during infancy are an emerging issue because of their potential link to metabolic health disorders in later life. Moreover, prolonged breast-feeding appears to be associated with a lower risk of obesity than formula feeding. Human milk is a source of various hormones and growth factors, namely adipokines (leptin and adiponectin), ghrelin, resistin and obestatin, which are involved in food intake regulation and energy balance. These compounds are either not found in commercial milk formulas or their presence is still controversial. Diet-related differences during infancy in serum levels of factors involved in energy metabolism might explain anthropometric differences and also differences in dietary habits between breast-fed (BF) and formula-fed (FF) infants later in life, and may thus have long-term health consequences.
Raloff, Janet. "Hormones: Here's The Beef. (Cover Story)." Science News 161.1 (2002): 10. MasterFILE Premier. Web. 31 Oct. 2013.
-Discusses the effects of hormones used to fatten beef cattle in the United States on the environment. Idea that many of the hormones pass through the cattle into the water table;
-Many cattle are fed the same muscle-building androgens-usually testosterone surrogates-that some athletes consume. Other animals receive estrogens, the primary female sex hormones, or progestins, semiandrogenic agents that shut down a female's estrus cycle. Progestins fuel meat-building by freeing up resources that would have gone into the reproductive cycle.
-So far, almost all concern about this practice has focused on whether trace residues of these hormones in the meat have human-health consequences. But there's another way that these powerful agents can find their way into people and other animals. A substantial portion of the hormones literally passes through the cattle into their feces and ends up in the environment, where it can get into other food and drinking water.
Some scientists say that it's time to better manage livestock's hormone-laced waste stream, which has flowed unabated in North America for decades.
As much as anyone, John A. McLachlan knows what's been happening. He first became interested in livestock hormones in the early 1970s, when he learned that farmers were giving the synthetic hormone diethylstilbestrol (DES) to chickens and cattle. This synthetic estrogen chemically castrates male animals, enabling them to grow faster. At the time, McLachlan's own studies at the National Institute of Environmental Health Sciences (NIEHS) used animal models to investigate why DES fostered the development of cancer in daughters of women treated to avoid miscarriages.
Hormone Deficiency and Growth Hormone Neurosecretory Dysfunction.
Spiliotis, Bessie E., et al. "Combined Growth Hormone-Releasing Hormone And Growth Hormone-Releasing Peptide-6 Test For The Evaluation Of Growth Hormone Secretion In Children With Growth Hormone Deficiency And Growth Hormone Neurosecretory Dysfunction." Hormone Research 70.4 (2008): 215-223. Academic Search Complete. Web. 31 Oct. 2013.
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Fifty children, aged 4.0–16.5 years, who were consecutively
referred to the Division of Pediatric Endocrinology and Diabetes
over a 3-year period because of severe growth delay (height
–2 SD and height velocity ! –2 SD) were included in the study
( table 1 ). Thirty-five (70%) had a peak GH response of ! 10 g/l
after provocation with levo-dopa and clonidine (classic GH deficiency;
GHD). The remaining 15 (30%) had a peak GH response
to pharmacologic provocation greater than 10 g/l but subnormal
spontaneous 24-hour GH secretion [low mean 24-hour GH concentrations
and low 24-hour GH secretory rates as compared to
their matched controls according to their sex and Tanner stages
( table 2 ), GH neurosecretory dysfunction, GHND] [9, 10, 13] . The
patients’ heights prior to treatment are given below in table 4 . All
patients had isolated GH deficiency.
Authors:Mendez R ; Grissom M
Mendez, Robert, and Maureen Grissom. "Disorders Of Childhood Growth And Development: Childhood Obesity." FP Essentials 410.(2013): 20-24. MEDLINE with Full Text. Web. 5 Nov. 2013.
Obesity is a multifactorial condition with syndromic and nonsyndromic variants. Genetic, social, ethnic, endocrinologic, and behavioral issues are all potential etiologic factors. Preventive efforts should begin with monitoring from birth and include breastfeeding until age 6 months, avoiding juices, and promoting fruit and vegetable consumption and adequate exercise. Childhood obesity is diagnosed based on body mass index; a child is considered overweight at the 85th to 95th percentiles and obese at or above the 95th percentile. After obesity is diagnosed, testing should include blood pressure levels, fasting lipid profile, diabetes screening, and liver function tests. The physician should obtain a detailed history of the physical activity level and food intake and assess possible complications of obesity, including depression and hypertension, annually.
Source 5:Hormonal Regulation of Farm animal Growth.
| Hossner, K. L. (Kim L.): |
| Hormonal regulation of farm animal growth[electronic resource] / |
| Wallingford, Oxfordshire, UK ; Cambridge, MA : CABI Pub., c2005. |
| viii, 223 p. : ill. |
| |
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